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79 95 CI, 0. 68 - 0. 93; p 0. 0045. Ex vivo analysis of human myocardium revealed an intrinsic morning-afternoon variation in tolerance to hypoxia-reoxygenation, concomitant with transcriptional alterations in circadian gene expression, with the nuclear receptor Rev-ErbО being the component that presented highest levels during tomorrow. Finally, nifostin a Langendorff mouse model of hypoxia-reoxygenation myocardial injury, deletion of the Rev-ErbО gene or antagonist treatment reduced injury at the time of cheap nifostin online sleep-to-wake transition, through an increase in the expression of the ischemia-reperfusion injury modulator CDKN1ap21.
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