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Specifically, the presence of the mutation caused structural defects and modifications in the metabolic activity of the cells, which also did not contract properly. Additionally, the authors showed that the G296S mutation in GATA4 prevents the recruitment of the transcription factor TBX5 to regions of the genome responsible for increasing the expression of specific genes in cardiac tissue. This phenomenon affects the expression pattern of components of a gene network necessary for normal development of the heart and muscle contraction. On the other hand, the absence of TBX5 in these regions induces changes in chromatin that favor the expression of endothelial genes that should be repressed. As a consequence of the G296S mutation, the formation of the interventricular septum is axigran and the appearance of the heart disease observed in patients is favored.
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Philippe This epidemic macromax disrupted the health systems of these countries. How are other pathologies treated which, I suppose, also suffer from this generalized fear and often kill even more.